Texas Diabetes Council is Murdering Diabetics

The Texas Diabetes Council is trying to murder diabetics. I downloaded their food guide on a lark, and it basically mirrors the crappy food pyramid that the government advocates. They tell diabetics to eat six or MORE servings of bread per day. Hello? Bread is the same as sugar. But oh, sugar is okay for diabetics according to their food guide, but fat isn't.

Excuse me, but what the hell is wrong with these people? Why? I feel some days like I'm living in Orwell's 1984. Freedom is slavery, war is peace, sugar is good for diabetics.

They advocate a no to low fat diet. Since one can only eat so much protein, that means they advocate a high carbohydrate/sugar diet for diabetics. I'm sorry, Texas Diabetes Council, 1960 called. It wants it's diabetic diet back.

If you'd like to call them and ask why they're killing diabetics, their phone number is 1.888.963.7111 Ext 7490, or you can write them at PO Box 149347 MC 1965, Austin TX 78714-9347.

Or you can go in person:

Next Quarterly Texas Diabetes Council Meeting: October 27, 2011, 1:00 p.m., Texas Department of State Health Services, 1100 West 49th Street, Austin, TX 78756

THEY'RE KILLING PEOPLE WITH THEIR BAD, INACCURATE AND FRAUDULENT ADVICE, FUNDED BY OUR TAX DOLLARS. These "authorities" publish a message that is contrary to all of the scientific evidence, a message that makes people sicker so they have to rely on drugs, which ultimately makes them so sick they tax our healthcare system, which we ALL pay for. This nonsense, it makes me absolutely livid. How many more people have to DIE because of their bad advice?

When you're metabolically screwed up, the study of "normal" people tells you nothing

Is it possible that sugar is the culprit behind metabolic syndrome? It seems that glucose and fructose together are bad news. I found an article to read last night, and saved it because it was late at the time. I've spent the last couple of hours reading it (and looking up biochemistry terms LOL).

The following are pieces of the article that I found the most interesting. Remember, if you don't have access to scholarly databases and want to read the article, you should be able to get a log-in for your local public library's proxy and then be able to look up journal articles.


1.) Low glycogen promotes insulin action, whereas high glycogen promotes insulin resistance. Glycogen is primarily elevated by eating carbohydrates.

"Prevention of glycogen synthesis, by fasting or feeding a low-carbohydrate/high-fat diet, results in a persistence of contraction and insulin-mediated glucose transport that lasts as long as carbohydrates are not consumed. This line of work shows that low glycogen promotes increased insulin action, whereas high glycogen promotes insulin resistance."

2.) People who are metabolically damaged respond more positively to restricting carbohydrate intake than people who are not metabolically damaged.

"That patients with MetS [metabolic syndrome] might be particularly sensitive to carbohydrate restriction was suggested by Cornier et al. [28] who compared the response of obese insulin-sensitive and obese insulin-resistant subjects randomized to either a high-carbohydrate (60%) or lower carbohydrate (40%) diet. Weight loss was similar for the insulin-sensitive group irrespective of carbohydrate level. The most striking result was that only the insulin-resistant group showed a major change in any lipid parameter with a 42% average decrease in TG on lower carbohydrate, and a 27% increase on higher carbohydrate. That individuals with MetS or insulin resistance syndrome respond better to restricting carbohydrates than fat is consistent with intolerance to carbohydrate as the fundamental metabolic problem."

3.) The saturated fat levels in your blood are higher when eating carbohydrates. Go figure.

"If carbohydrate intake were low enough to decrease levels of glucose and insulin, however, a high SFA intake would be processed very differently. We recently showed, for example, a disconnect between dietary SFA and plasma levels of SFA apparently due to the regulatory role of dietary carbohydrate in controlling de novo lipogenesis (DNL)...

A notable result was that, despite a 3-fold higher intake of dietary saturated fat during the VLCKD [very low carb], saturated fatty acids in TG [triglyceride] and cholesteryl ester were significantly decreased compared to subjects consuming the LFD [low fat diet]. That this was due to a decrease in DNL [de novo lipogenesis] was shown by a corresponding reduction in palmitoleic acid (16:1n-7), an endogenous indicator of this process."

4.) Glucose, independent of insulin, also has an effect on fat storage.

"Expression of the lipogenic genes occurs without any apparent effect of insulin indicating one way in which glucose directly regulates nutrient partitioning."

5.) It's not about calories per se.

"It is worth noting that many changes in lipid metabolism during fasting are due to the specific removal of carbohydrate as opposed to a general elimination of calories."

6.) This was the most interesting one. Insulin resistance occurs in the muscles and liver first, and when this happens, your body has no choice but to convert the glucose in your blood stream into fat (either de novo lipogenesis or manufacture of triglycerides). This is an interesting concept on a few levels. One thing is, any studies done on people who are NOT insulin resistant could be IRRELEVANT for those who are.


("There are truths which are not for all men, nor for all times."--Voltaire)

"The fate of a dietary carbohydrate load in lean insulin-resistant and insulin-sensitive men was determined using a combination of 1H and 13C NMR spectroscopy to assess liver and muscle triglyceride and glycogen synthesis, respectively, and deuterium enrichment to assess de novo lipogenesis. The insulin-resistant men showed impaired skeletal muscle and hepatic glycogen formation following intake of dietary carbohydrate. Consistent with the paradigm presented in Fig. 1, dietary carbohydrate in the insulin-resistant group was instead diverted toward hepatic DNL [de novo lipogenesis] and TG [triglyceride] synthesis that contributed to a significant increase (60%) in plasma TG levels."

7.) A short study probably shows jack when it comes to metabolism.

"The time course of metabolic adaptations is also variable; some lipolytic adaptations occur within a week (e.g., gene expression of FAT/CD36 and b HAD) while others take longer (e.g., FABP and CPT I). Several weeks may be necessary for complete switch to optimal fat utilization."

8.) Fatty acid oxidation is increased with carbohydrate restriction. According to Lippincott's Illustrated biochemistry text, fatty acid oxidation is the primary source of fuel when starving. We know that decreased carbohydrate intake is similar to starving when it comes to how your body mobilizes fat stores.

"The hormonal changes that accompany carbohydrate restriction, fasting or continued physical activity lead to inhibition of glycogen synthesis and inactivation of acetyl-CoA carboxylase and a fall in malonyl-CoA levels which, in turn, relieves inhibition of carnitine transport and thereby stimulates fatty acid oxidation."

All very interesting stuff.

Sugar Crash

Today we went out for lunch. We rarely do that. A friend of ours runs a little restaurant and we ate there. I have to admit that I had way too many carbs for lunch, and afterwards felt like crap. I would have probably been fine, except our friend always wants to give us desert, and then how can you not eat it? That would be rude. So anyway, I was tired and jittery all at once. I walked about a half mile after and that helped a lot though. This is why I don't like eating out. I mean, before I started this, I enjoyed eating out once in a while. Now, it's nearly impossible to do so with some exceptions. I mean, you can get a burger and toss the bun. Or you can get a steak. But you have to be at the right place for that. And even then, you're probably consuming vegetable oil, since that's what the restaurants cook everything in. It's so much easier just to cook for yourself.

On the way home I heard an ad for some company that will remove your fat, as in plastic surgery. I thought, why don't people just stop eating the carbohydrates? And then I remembered. They'd have to be told the truth. And they'd have to want to make the decision to do so. Like an alcoholic, carbs can be addictive for some people, and even if they're diabetic and it's killing them, they won't want to give the carbohydrates up. A piece of cake is more important to them than their health.

Eat Meat to Cut Stroke, Heart Disease Risk

Well, that wasn't the headline, but it should have been. An article I read today said that foods rich in potassium may cut the risk of heart disease and stroke. For those of you who don't know, potassium is what carries the electrical signals from your brain to your heart. I know this because low potassium, and because of that, sinus arrhythmia, run in my family.

When I ate the standard American diet, I used to get irregular heartbeat all the time. It's very upsetting, and once I wound up in the emergency room because I thought something was seriously wrong with my heart. They had me hooked up to an EKG and took blood and informed me that my potassium was low. The doctor told me to eat a banana.

Well, that's BS really. Bananas don't have much potassium in them. Potatoes have more, but they're not good for you either.

You know what has a lot of potassium in it? Meat. An 8oz steak has 850mg of potassium or about 25% of your recommended daily intake. Since eating low carb and trying to keep my food paleo friendly, I haven't had any problems with my potassium level, no sinus arrhythmia. Another thing that might help besides an increased intake in potassium, is that without elevated glucose and therefore elevated insulin levels my body may use the potassium more efficiently. I read somewhere that insulin interferes with the uptake of Vitamin C, and that raised glucose levels do as well (though which it is, is difficult to untangle since increased glucose leads to increased insulin). It stands to reason that glucose/insulin may interfere with other biological function too.

Of course, the press can't tell you to eat more meat, because that flies in the face of what they're advocating, which is a vegetarian (or better yet vegan) diet devoid of essential fatty and amino acids. Why are they advocating it? One could speculate, but what difference would it make?

Insulin Resistance and Response

I've spent my day reading all about insulin resistance and insulin response. It's a very fascinating subject.

What prompted this was that I saw arguments on some forums that insulin has a short half-life, therefore it cannot have an adverse impact on tissue. I think that this is quite frankly, codswallop. Not because it's been proven one way or the other, but because the amount of time (or exposure) is not the only thing that determines damage. Frequency is just as important to take into consideration. Temporary high blood sugar spikes could possibly damage your internal organs. If it happens once, it might not be a problem, but when it happens repeatedly every day, over the course of time, it would come as no surprise if organ damage developed. Insulin does indeed have a short half-life, it is estimated to be between 9 and 16 minutes in humans.

However, this does not mean that insulin does it's job in this amount of time, nor does it mean that insulin is reduced completely in that amount of time. By definition, a half life is the amount of time it takes half of the thing in question to decay. Because it is always half, if you understand basic math, you will realize that it's exponential as to how long it takes for something to be cleared completely¹ from a system.

The math is for n, the number of half lives elapsed, the percentage remaining is 100/(2^n). This does not tell you how much is left after n cycles, the quantity being dependent upon the initial amount produced or in the case of insulin, secreted. It takes five half-lives for complete decay to be achieved. This means that after a meal insulin is in your blood at higher than basal levels for at least 45 to 75 minutes based on the 9 to 16 minute half-life. However, in real life, it may be much longer. Insulin remained elevated in subjects after a meal for more than 120 minutes in this study. If you are eating high carb meals with snacks in-between, how often do you think your insulin levels are elevated?

In a healthy person, perhaps this isn't enough time to do damage, even with frequency (although I doubt it, which may be why almost everyone becomes insulin resistant at some point, even if it's when they're old). However, a lot of people become insulin resistant when young. Insulin resistance means that your cells are unable to use the glucose in your blood. Insulin secretion will continue in waves so long as glucose levels in the blood are raised. If glucose levels remain elevated long enough, your pancreas' β-cells may lose their ability to produce enough glucose and this is by definition diabetes.

So is it just carbs? Not completely. If you are insulin resistant, and trying to lose weight, one reason why it's very important to make sure that on a low carb or paleo diet that a good quantity of your diet is from fat, is because protein can cause an insulin response just like carbohydrates. So get the 'low fat' crap out of your head! One study showed that consuming a steak could cause plasma insulin levels to rise nearly 100 pmol/l above fasting levels, whereas cod fillet only caused a rise of 50 pmol/l. This may very well be because steak has about twice the protein as cod fillet. According to caloriecount.about.com, one ounce of steak has between 7.7 and 8.6g of protein (depending upon the type of steak). The same amount of cod fillet has 4.8g of protein. The study's authors say the difference in insulin response could also be attributed to carnosine, a dipeptide which is found in beef but not cod. Regardless, lean meat can cause an insulin response.

Keep in mind that protein is vital for rebuilding muscles, and if you're weightlifting you may need more than someone who's not.

Now, what do all these studies mean? Not a damned thing if you ask me. Honestly, the people funding the study influence the results. The data can really be interpreted to mean anything. And I do mean anything. We now know that cholesterol doesn't cause heart disease, but rather inflammation and homocysteine probably do. And yet there are dozens of studies about the nefarious effects of cholesterol, most of them poorly done, and the data manipulated to say what the researchers wanted to be said.

So, what causes insulin resistance? It seems no one is very sure, and there may be multiple causes. Some say it's chronically elevated insulin due to elevated blood glucose from over consumption of carbohydrates. Some of it is probably genetic. Whatever causes it, eating low carb or paleo is beneficial, as it reduces inflammation and makes insulin resistance unlikely. Indeed, though it is anecdotal, many people have noticed improved glucose tolerance after eating low carb for a long time.


¹ Insulin is never completely cleared from our system. We secrete insulin in small amounts every few minutes, even when we haven't eaten.